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in the tissues, O 2 is unloaded from Hb that undergoes a spatial rearrangement of the globin chains, allowing the entry of 2,3-diphosphoglycerate (2,3-DPG) which diminishes O 2 affinity [1, 2]

In aged and senescent cells, the balance shifts decisively toward repression: the hTERT promoter becomes hypermethylated, and the surrounding chromatin adopts a condensed, transcriptionally inactive configuration

Studies in rat models demonstrate that spironolactone can prevent histological and functional alterations caused by doxorubicin, including myocardial fibrosis and elevated cardiac enzymes [170]