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In summary, semaglutide-induced nausea and vomiting are thought to result from a combination of delayed gastric emptying and direct activation of central emetic pathways [60, 62, 63]
And it acts on appetite centers in your brain, reducing hunger and increasing feelings of satisfaction after meals
The question is not whether the mechanism makes sense but whether clinical trials will confirm the magnitude of benefit suggested by preclinical and early observational data
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