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However, a high proportion of people with T2D discontinued GLP-1 RA treatments despite a low proportion of people with GI AEs
It also stimulates the formation of new blood vessels (angiogenesis) through the VEGF pathway, which is how injured tissue gets the nutrients it needs to rebuild
ER stress can further tighten ER-mitochondria communication and augment Ca2+ dependent NLRP3 responses together with remodeling of MAM-associated factors and mitochondrial dynamics, reinforcing that inflammatory state is regulated at contact sites (33)
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