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Moreover, the translation of human genetic and preclinical findings in ASD can provide mechanistic insights and clues to rescue different behavioral and synaptic deficits in early preclinical models, which may serve as a basis for future clinical trials
Structural assignments were further supported by J-resolved and TOCSY spectra and validated using carbon chemical shifts from HSQC spectra
Hyperglycaemia progressively impairs these transporters, accelerating beta-cell apoptosis and microvascular complications
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