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In the selenite-induced cataract model, the extract likely mitigated oxidative stress by scavenging reactive oxygen species, thereby preventing protein damage and maintaining the levels of essential lens proteins such as -A crystallin and aquaporin 0

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Instead, it emerges from the coordinated interaction of metabolic branch competition, spatiotemporal regulation, hormone crosstalk, and multilayered transcriptional control

Dose above the clinically studied range does not meaningfully speed up the timeline