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By targeting key molecular pathways such as Wnt/-catenin, SMAD, PI3K/AKT, and MAPK/ERK, these therapies can modulate gene expression, cell signaling, and protein synthesis to optimize healing outcomes
The mechanism centers on several overlapping actions: Angiogenesis activation: BPC-157 upregulates VEGF (vascular endothelial growth factor), promoting the formation of new blood vessels that supply oxygen and nutrients to damaged tissue
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