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When glucose was low, more exendin-4 entered the median eminence, showing that glucose levels themselves influence how much of the drug reaches the brain
Small intestine conditions such as celiac disease, Crohns disease, parasite or bacterial growth
In theory, this could make the transition smoother for GI effects related to GLP-1 and GIP, while the glucagon-mediated effects (thermogenesis, increased heart rate) would be entirely new
I mean, I'm fascinated by how mitochondria are perhaps transported between cells and around the body
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