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Additionally, it supports immune function, helping the body maintain balance against daily stressors and environmental challenges
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The observed reduction in GSSG levels reflects a compensatory regulation of redox balance, suggesting a potential self-protective mechanism that may play a crucial role in maintaining neurological homeostasis [48, 49]
Elevated levels of ferritin and transferrin, markers of iron dysregulation, have been observed in clinical ARDS cases, linking iron metabolism to disease severity ( Mitochondrial dysfunction exacerbates iron-driven ferroptosis by impairing the organelles ability to sequester and regulate iron levels
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