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Both are biologically plausible, and both have limited supportive human evidence
The profibrotic mechanism of ferroptosis is closely related to the TGF-/Smad pathway, and targeting ferroptosis and increasing GPX4 expression could be an effective strategy for treating CKD
For dual agonists: because they are stronger, the sideeffect profile may be similar or somewhat amplified
Its also generally accepted that they work by targeting the brains reward systems, which could explain why they may help with addiction
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