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In addition, cholesterol in the tumor environment can upregulate the expression of CD36 in tumor-infiltrating CD8 + T cells, which can increase the ingestion of fatty acids, promote lipid peroxidation and ferroptosis as well, thus compromising the anti-tumor function of T cells and promoting tumor progression [155]
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Chondrocyte senescence (Zhu et al., 2021): Selectively removed senescent cells from human chondrocyte cultures, with implications for cartilage repair
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