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Full-text article Neelakantan H, Brightwell CR, Graber TG, et al
Beyond its antioxidant role, Glutathione is central to Phase II hepatic detoxification research, where it acts as a conjugate for electrophilic xenobiotics and reactive metabolites via glutathione S-transferase (GST) enzymes, facilitating their excretion
33,41 Accordingly, the present study confirmed the critical role of inflammation in the mechanism of DOX-induced nephrotoxicity, as demonstrated by a significant increase in inflammatory markers, including serum CRP, NLR, MLR, and renal tissue TNF- and IL-1 in the DOX control group
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