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An overview of the stomach-stress-organoprotection hypothesis and beneficial effects of BPC

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2.5 Core role of mitochondrial dysfunction in ferroptosis In recent years, a growing body of research has revealed that mitochondria play a key regulatory role in the occurrence and development of ferroptosis (114, 115)

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We hypothesized that there might be a transient lack of metabolically available antioxidants that could induce a suboptimal quality of ovulation and, eventually, of (or some) embryos

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