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This pharmacologic feature is directly relevant to hepatic impairment because the drug's efficacy does not depend on liver enzyme function for activation
global statistical test 0.08, P = .70)
Diabetes Obes Metab 24:94105 Funding This work was supported by the funding from The Foundation of Hope for Research and Treatment of Mental Illness and in part by the Bowles Center for Alcohol Studies
The metabolism of BA after MBS is not fully known, but it is recognised that they act on cells in the intestinal wall by activating the farnesoid x receptor (FXR) and Takeda G protein-coupled receptor 5 (TGR5), which potentiates the release of GLP-1 [12]
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