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These mechanisms connect to physiological outcomes (faster wound healing, improved tendon repair, protection of the gastrointestinal lining), but current understanding is based largely on laboratory and animal studies, with limited confirmation in controlled human trials
demonstrated that sickle cell anemia correlates with elevated eryptosis markers, such as PS externalization, increased ROS within RBCs, and elevated intracellular calcium levels
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Differential toxicity of TAR DNA-binding protein 43 isoforms depends on their submitochondrial localization in neuronal cells
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