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PKA has been shown to phosphorylate -catenin at two specific locations, Ser552 and Ser675 (Hino et al
[9] [11] While these mechanisms contribute to improved glycemic control and weight loss, they also explain why gastrointestinal side effects are among the most commonly reported adverse reactions
The medication works primarily by mimicking the naturally occurring GLP-1 hormone, which enhances insulin secretion, suppresses glucagon release, slows gastric emptying, and reduces appetite through central nervous system pathways
The glucagon receptor component influences energy expenditure and fatty acid oxidation
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