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After the macrophages ingest MSU through the phagocytic pathway, NLRP3 inflammasome assembly and activation are triggered, and the generated complex consists of NLRP3, the adaptor protein apoptosis-related cyclin-like protein (ASC), and caspase-1 (53)

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We first validated the buffering phenotype in the follow-up studies, in which SLC25A19 exhibited milder growth fitness defect compared to the control cells in the presence of complex III inhibitor antimycin, complex I inhibitor piericidin and complex V inhibitor oligomycin than in the regular culture condition (Supplementary Fig

Social stress induces neurovascular pathology promoting depression