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The rationale is elegant: because GLP-1 receptors are enriched in metabolically active brain regions, using GLP-1 as a delivery vehicle allows estrogen to be directed preferentially toward the central nervous system, potentially enhancing its neuroprotective effects while limiting systemic hormonal exposure
On April 30, 2026, the U.S
J Cell Physiol 181: 470-478, 1999
[6] Beyond pigmentary effects, the gray-market literature also describes nausea and facial flushing, spontaneous penile erection (priapism, an MC4R-mediated effect), and occasional systemic reactions such as rhabdomyolysis
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