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Semaglutide works by slowing gastric emptying and signaling fullness to your brainbut this metabolic shift sometimes triggers temporary fatigue
Although this increases cytosolic gluconeogenesis, it is coupled with a crucial secondary effect: calcium transfer to mitochondria activates key tricarboxylic acid (TCA) cycle dehydrogenases, boosting mitochondrial energy expenditure and reducing ectopic lipid accumulation [50]
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Minimal Downtime: Most individuals can resume normal activities shortly after treatment
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