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2012.DOI Pickart L., Vasquez-Soltero J., Margolina A
The activity of CPT1C is distinct from those of CPT1A and CPT1B in that it does not act on the same types of fatty acyl-CoAs that are substrates for the latter two enzymes, nor does it participate in the mobilization of fatty acids into the mitochondria
However, in Sprague Dawley rats, GLP-1 monotherapy significantly increased kaolin intake compared with vehicle-treated rats and rats treated with MK-801 (Extended Data Fig
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