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on the contrary, fission allows mitochondria to constantly divide and is mainly related to apoptosis, facilitating the segregation of mtDNA in mitosis and eliminating defective mitochondria ( The anti-apoptotic protein Bcl-2 homologous antagonist/killer (BCL-2), overexpressed in some subtypes of B-cell lymphomas, regulates mitochondrial function and mediates therapeutic resistance by inhibiting the oligomerization of the pro-apoptotic Bcl-2 homologous antagonist/killer (Bak) and Bcl-2-associated X protein (Bax) proteins and by facilitating the import of GSH and complex IV subunits to mitochondria (Mattes et al., 2019)
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