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Beyond defining a mitochondrial mechanism for fuel switching, this work provides a conceptual landscape for future investigation of metabolic adaptation across diverse physiological contexts, including exercise, distinct fasting paradigms, and hormone-driven metabolic regulation, and offers new insight into the determinants of responsiveness to GLP1R agonist-based anti-obesity therapies
ROS also damage nucleic acids, particularly guanine bases, resulting in eight-oxoguanine formation, DNA strand breaks, and telomere shortening (Bloemberg and Quadrilatero, 2019)
10.1073/pnas.1001006107 110 Matoba S., Kang J
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