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Metabolic Fate: BPC-157 is primarily metabolized in the liver, where it is broken down into smaller peptide fragments and individual amino acids that enter normal metabolic pathways
According to the results from in vitro and in vivo studies, tanshinone IIA pretreatment protects the liver from APAP-induced liver injury by activating Nrf2 and increasing the mRNA and protein levels of the Nrf2 target genes glutamate-cysteine ligase catalytic subunit (GCLC), NQO1, and HO-1 (Wang et al., 2016)
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