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59 These findings suggest that vesicle-mediated signaling may either amplify or restrain ferroptosis depending on cargo composition and uptake efficiency, highlighting extracellular vesicles as bidirectional regulators of ferroptotic susceptibility rather than unidirectional death signals

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study documented significantly accelerated tendon-to-bone healing in rat rotator cuff models compared to untreated controls, with VEGF pathway upregulation as a proposed mechanism [1]

Anholm C, Kumarathurai P, Pedersen LR, Samkani A, Walzem RL, Nielsen OW, Kristiansen OP, Fenger M, Madsbad S, Sajadieh A