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Eve Roberts, writing in the AASLD Practice Guidelines for Wilson Disease , emphasized that "any exogenous copper load in a patient with compromised biliary excretion has the potential to accelerate hepatic copper accumulation" [2]
Polymorphisms on this chromosome, as well as endogenous androgen effects, have been linked to TMPRSS2 gene transcription and activation of the antiviral enzyme ACE2, which facilitates viral penetration in target cells [12]
They function as signaling molecules in the body, telling specific cells and systems what to do
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