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Their complementary mechanisms (signaling amplification via BPC 157 + cell migration facilitation via TB 500) provide a scientific rationale for combined investigation

UA administration to mice before the onset of clinical EAE may protect the integrity of the BBB from changes in ONOO -induced permeability, thus minimizing the infiltration of inflammatory cells and other pathogenic cells and factors and the resulting pathology [193](Fig

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doi:10.1210/jc.2003-030965