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Our review intends to elucidate recent discoveries regarding the pathogenetic role of the most studied RCD, such as apoptosis, necroptosis, pyroptosis, ferroptosis and autophagy, in DKD, and provide a theoretical foundation for future investigations as well as targeted drug design
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(iii) inhibiting lipolysis, decreasing plasma fatty acids, and reducing liver glucose output
It should be noted, however, that the Grx activity of the enzyme was sustained without the C-terminal motif, as shown here and previously [11]
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