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By inhibiting NNMT, the body maintains higher NAD levels, which in turn activate SIRT1, a longevity-associated gene, which may improve mitochondrial activity, fat oxidation, insulin sensitivity, and by overall energy balance
It works by mimicking two gut hormones, glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) receptor which helps to lower blood sugar levels and reduce appetite
Research has also explored its interaction with pathways associated with: Fibrosis-related signaling Oxidative-stress regulation Chronic inflammationrelated pathways Cellular-maintenance and adaptive-response mechanisms Because of its broad interaction with regenerative signaling pathways, GHK-Cu is frequently investigated in skin-integrity, healthy-aging, tissue-maintenance, and recovery-focused research models
Clinical Research and Evidence While large-scale human trials are still limited, the body of research on BPC-157 peptide paints a compelling picture of its therapeutic potential, especially in conditions that mirror the challenges of chronic Lyme disease
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