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Thus, we set out to determine, first, whether Kat6a +/ mice could be used as a model for ARTHS with respect to effects on histone acetylation and cognitive and social functions and, second, whether treatment aimed to increase histone acetylation levels might ameliorate any learning, memory, or social dysfunction that might be present

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The diabetes drug liraglutide reverses cognitive impairment in mice and attenuates insulin receptor and synaptic pathology in a non-human primate model of alzheimers disease

These two peptides operate through distinct but complementary receptor pathways GHRH receptors (CJC-1295) and ghrelin receptors (Ipamorelin)