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Amylin is co-located with insulin in secretory granules and co-secreted with insulin by pancreatic beta cells in response to food intake

However, in conventional DIO mouse models, blocking GLP-1 receptor activity abolishes tirzepatides efficacy, while blocking GIP receptor activity reportedly has minimal impact on its effects on insulin secretion or blood glucose

This suggests that the improvements in metabolic parameters were primarily driven by the modulation of lipid and glucose metabolism, rather than the suppression of inflammation

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