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Vitamin B12 deficiency results in megaloblastic anemia, GI lesions, and neurologic damage (which begins with an inability to produce myelin and is followed by gradual degeneration of the axon and nerve head)
Current availability is confined to the research-chemical market, without regulatory oversight or established human safety data
In summary, making B12 therapy better is a detailed job that focuses on the patient
Supporting behaviors significantly impact results, potentially more than minor dose adjustments or timing optimization
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