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This dual-action mechanism gives patients an additional boost by improving fat metabolism and regulating insulin even further
A., Flatt, P
While the oral bioavailability of aspirin is 40-50%, clinical studies indicate that the majority of aspirin's irreversible inhibition of platelet COX-1 takes place in the presystemic (portal) circulation, resulting in aspirin's antiplatelet effect being largely independent of systemic bioavailability (Pedersen & FitzGerald, 1984
This means lighter individuals experience higher peak concentrations relative to their body weight, which may explain why they often report more intense side effects and a stronger wearing-off effect
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