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By regulating glutathione S-transferase (GST) and intracellular glutathione (GSH) levels, Nrf2 controls the level of ROS in the cell (Kolls, 2006
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NPs-induced downregulation of endothelial nitric oxide synthase (eNOS) expression, causing impaired endothelium-dependent vasorelaxation and promoting reactive oxygen species creation in endothelial cells, which was associated with high NADPH oxidase expression, with subsequent downregulation of Sirt1 expression [76]
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