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Neurological injuries
Such stress-associated -cell states are known to display impaired incretin responsiveness due to reduced GLP1R signaling and diminished cAMP pathway activation (Luo et al., 2013)[Gao], suggesting that inflammatory axes linked to dysbiosis may differentially affect vulnerable -cell subpopulations
The peptide's research profile has expanded from its origins in gastrointestinal cytoprotection, first described by Sikiric et al., to a broad range of tissue repair applications
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