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Altogether, these data suggest that the upregulation of GSH, rather than the dTTP pool, is responsible for avoiding oncogene-induced replication stress and less susceptibility to JMF4073 in T315I-Bcr-Abl-32D cells

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ZhuWGregoryJCOrgEBuffaJAGuptaNWangZet alGut microbial metabolite TMAO enhances platelet hyperreactivity and thrombosis risk

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