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3.2 Iron dysregulation in modulating ferroptosis susceptibility Iron overload, as a key inducer of ferroptosis, disrupts cellular iron homeostasis by interfering with critical components of iron metabolism at both transcriptional and post-transcriptional levels, thereby significantly increasing cellular susceptibility to ferroptosis (59)

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Collectively, these findings suggest that behavioral recovery in ASD models involves mechanisms that are more complex than the mere normalization of oxidative stress markers, requiring the integration of multiple neural circuits and pathophysiological processes