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Chronic non-communicable diseases (NCDs), including cardiovascular disease, Type 2 diabetes, obesity, metabolic syndrome, and metabolic-associated fatty liver disease, continue to rise worldwide 1
However, drastic redistributions of MDM2 from p53 to the IGF-1R can probably only be achieved after experimental modulations, like inhibition of p53 expression by antisense strategies, etc, and do not likely occur in this manner in a physiologic context
The ORIGIN Trial Investigators
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